Melatonin modulates the fetal cardiovascular defense response to acute hypoxia

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Melatonin modulates the fetal cardiovascular defense response to acute hypoxia

Experimental studies in animal models supporting protective effects on the fetus of melatonin in adverse pregnancy have prompted clinical trials in human pregnancy complicated by fetal growth restriction. However, the effects of melatonin on the fetal defense to acute hypoxia, such as that which may occur during labor, remain unknown. This translational study tested the hypothesis, in vivo, tha...

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In complicated pregnancy, fetal hypoxemia rarely occurs in isolation but is often accompanied by fetal acidemia. There is growing clinical concern about the combined effects of fetal hypoxemia and fetal acidemia on neonatal outcome. However, the effects on the fetal defense responses to acute hypoxemia during fetal acidemia are not well understood. This study tested the hypothesis that fetal ac...

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Fetal stress. Focus on "effects of acute acidemia on the fetal cardiovascular defense to acute hypoxemia" by Thakor and Giussani.

WHAT CONSTITUTES FETAL stress, and what are the implications of fetal stress in the course of fetal development? Thakor and Giussani (17) address a fundamental component of the fetal stress response: acidemia and it role as a modulator of the hypothalamus-pituitary-adrenal (HPA) axis responsiveness to hypoxia. Fetal stress responsiveness has long been studied from the perspective of fetal survi...

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Acute cardiovascular response to isocapnic hypoxia. II. Model validation.

The role of the different mechanisms involved in the cardiovascular response to hypoxia [chemoreceptors, baroreceptors, lung stretch receptors, and central nervous system (CNS) hypoxic response] is analyzed in different physiological conditions by means of a mathematical model. The results reveal the following: 1) The model is able to reproduce the cardiovascular response to hypoxia very well b...

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ژورنال

عنوان ژورنال: Journal of Pineal Research

سال: 2015

ISSN: 0742-3098,1600-079X

DOI: 10.1111/jpi.12242